As a major intracellular antioxidant, it directly neutralises reactive oxygen species (ROS) and free radicals that accumulate during normal metabolism and are elevated in fatty liver disease

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This effect is dependent on phospholipase C activation and subsequent calcium release from IP 3 -sensitive intracellular stores [351]
All LOX isoforms promote PUFA peroxidation, and their knockdown has been consistently shown to attenuate ferroptosis (43)